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Rapamycin Review Article

 

In the years since, rapamycin has been adapted for numerous uses. Like penicillin, itís a biological agent, so it canít be patented, although derivatives of it can. Itís now used routinely as a coating on cardiac stents to prevent scarring and blocking. Derivatives of rapamycin have been approved for use against certain kidney, lung, and breast cancers. That may be just the beginning. Over the past decade, itís shown promise as a drug that not only can extend life by delaying the onset of aging-related diseases such as cancer, heart disease, and Alzheimerís disease, but also postpone the effects of normal aging. With an eye toward changing the way millions grow older, Novartis, the $260 billion Swiss pharmaceutical giant, has begun taking the first steps to position a version of rapamycin as the first true anti-aging drug.

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Rapamycin works at a fundamental level of cell biology. In the early 1990s, scientists at Novartisís predecessor, Sandoz, discovered that a rapamycin molecule inhibits a key cellular pathway regulating growth and metabolism. This pathway was eventually dubbed ďtarget of rapamycin,Ē or TOR, and itís found in everything from yeast to humans (itís known as mTOR in mammals).

MTOR is like the circuit breaker in a factory: When itís activated, the cell grows and divides, consuming nutrients and producing proteins. When mTOR is turned down, the ďfactoryĒ switches into more of a conservation mode, as the cell cleans house and recycles old proteins via a process called autophagy. One reason caloric restriction extends life span in animals, researchers believe, is because it slows down this mTOR pathway and cranks up autophagy. Rapamycin does the same thing, only without the gnawing hunger.


 

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